A new enzyme has been discovered that can trigger the treatment of celiac disease. Researchers at the Stanford University have discovered how a disease-associated protein gets inactivated opening the door to possible new treatments for the disease. Celiac disease is an autoimmune disorder that affects by some estimates nearly 1 in 100 people. Celiac disease symptoms are triggered by gluten a protein found in wheat and related plants but gluten doesn't act alone to cause the digestive symptoms that patients suffer. Rather gluten induces an overactive immune response when it's modified by the enzyme transglutaminase 2 or TG2 in the small intestine.